Inside every vial

Proof you can read, down to the sequence.

You deserve to know exactly what’s in every vial. So we characterize everything - how each peptide works at the molecular level, its exact sequence and molecular weight, and the published research behind it. No mystery blends. No marketing fog. Just documented, peer-sourced science you can verify yourself - lab-tested and confirmed down to the amino acid.

Recovery & Repair Energy & Longevity Metabolic & Incretin Signaling Blends Supplies

IdentitySequence & CAS documented

PurityCOA on every vial

TestingThird-party verified

ProvenanceMade in the USA, lot-traceable

Research category

Recovery & Repair

03 peptides

BPC-157The bounce-back peptide

Gly-Glu-Pro-Pro-Pro-Gly-Lys-Pro-Ala-Asp-Asp-Ala-Gly-Leu-Val · MW 1419.53 g/mol · CAS 137525-88-5 · C62H98N16O22

Push hard, recover harder. BPC-157 is the recovery favorite for people who refuse to let nagging aches slow them down - lab-tested for purity, COA on every vial.

Mechanism of action

Upregulates growth-hormone receptor expression on tendon fibroblasts.
Modulates the nitric-oxide pathway via endothelial NOS, regulating vascular tone.
Upregulates VEGF expression, associated with microvascular formation in models.

Studied in the literature

Soft-tissue & musculoskeletal repair in animal tendon-transection models.
Gastrointestinal mucosal protection and anastomosis models.
Vascular endothelial recovery and collateral-vessel formation in vivo.

Selected citations

Chang, C. H., et al. (2014). Pentadecapeptide BPC 157 enhances the growth hormone receptor expression in tendon fibroblasts. Molecules. doi:10.3390/molecules191119066 Sikiric, P., Seiwerth, S., et al. (2018). Stable gastric pentadecapeptide BPC 157, G-protein coupled receptor, and angiogenesis. Current Pharmaceutical Design. doi:10.2174/1381612824666180608101119

TB-500Whole-body recovery, dialed in

Ac-Ser-Asp-Lys-Pro-Asp-Met-Ala-Glu-Ile-Glu-Lys-Phe-Asp-Lys-Ser-Lys-Leu-Lys-Lys-Thr-Glu-Thr-Gln-Glu-Lys-Asn-Pro-Leu-Pro-Ser-Lys-Glu-Thr-Ile-Glu-Gln-Glu-Lys-Gln-Ala-Gly-Glu-Ser · MW 4963.50 g/mol · CAS 77591-33-4 · C212H350N56O78S

When you want to feel limber, mobile, and ready again, TB-500 is the go-to for full-body recovery and flexibility - third-party verified, made in the USA, lot-traceable.

Mechanism of action

Binds monomeric G-actin, regulating cytoskeletal dynamics and cell motility.
Stimulates matrix metalloproteinases associated with endothelial migration.
Upregulates L-selectin expression linked to progenitor-cell homing.

Studied in the literature

Musculoskeletal and myocardial recovery / vascularization models.
Corneal and epithelial re-epithelialization models.
Dermal repair and keratinocyte migration studies.

Selected citations

Goldstein, A. L., et al. (2012). Thymosin beta 4: actin-sequestering peptide pathways in wound healing and tissue regeneration. Vitamins and Hormones. doi:10.1016/B978-0-12-398397-8.00010-9 Smart, N., et al. (2011). Thymosin beta 4 induces adult myocardial regeneration and neovascularization. Nature. doi:10.1038/nature10188

GHK-CuYour skin’s glow-up in a vial

Gly-His-Lys (copper-complexed) · MW 401.91 g/mol · CAS 89030-95-5 · C14H22CuN6O4

The copper peptide skincare obsessives swear by - for firmer, smoother, lit-from-within skin and that fresh, rested look - lab-tested for purity, COA on every vial.

Mechanism of action

Modulates transcription of collagen types I, III and IV in dermal fibroblasts.
Balances MMP / TIMP activity associated with clean ECM remodeling.
Tunes inflammatory cytokines (TNF-α, IL-1β) in tissue-culture models.

Studied in the literature

Epithelial remodeling and anti-aging tissue-culture models.
DNA-repair and epigenetic gene-expression studies.
Wound-healing and angiogenesis models.

Selected citations

Pickart, L., et al. (2015). GHK peptide as a natural modulator of multiple cellular pathways in skin regeneration. BioMed Research International. doi:10.1155/2015/648108 Pickart, L., & Margolina, A. (2018). Regenerative and protective actions of the GHK-Cu peptide in the light of new gene data. Cosmetics. doi:10.3390/cosmetics5020029

Research category

Energy & Longevity

04 peptides

CJC-1295 No DACWork with your body’s natural rhythm

CJC-1295 No DAC - HappyTides research vial

Tyr-D-Ala-Asp-Ala-Ile-Phe-Thr-Gln-Ser-Tyr-Arg-Lys-Val-Leu-Ala-Gln-Leu-Ser-Ala-Arg-Lys-Leu-Leu-Gln-Asp-Ile-Leu-Ser-Arg-NH2 · MW 3367.97 g/mol · CAS 863288-34-0 · C152H252N44O42

For people chasing deeper sleep, leaner composition, and that younger-you energy, CJC-1295 No DAC supports your body’s own growth-hormone rhythm - lab-tested purity, COA on every vial.

Mechanism of action

Binds GHRH receptors on pituitary somatotropes with high affinity.
Raises intracellular cAMP via adenylate-cyclase / GPCR signaling.
Mimics pulsatile GH release without DAC-mediated albumin binding.

Studied in the literature

Growth-hormone-axis stimulation without affecting cortisol / thyroid markers.
Adipose and somatotropic-axis signaling models.
Cellular and mitochondrial respiration studies via IGF-1 signaling.

Selected citations

Teichman, S. L., et al. (2006). Prolonged stimulation of growth hormone and IGF-I secretion by CJC-1295. J. Clinical Endocrinology & Metabolism. doi:10.1210/jc.2005-1536 Alba, M., et al. (2005). Once-daily administration of CJC-1295 maintains pulsatile GH secretion. Am. J. Physiology-Endocrinology and Metabolism. doi:10.1152/ajpendo.00092.2005

TesamorelinFor a leaner, sharper you

trans-3-hexenoyl-Tyr-Ala-Asp-Ala-Ile-Phe-Thr-Asn-Ser-Tyr-Arg-Lys-Val-Leu-Gly-Gln-Leu-Ser-Ala-Arg-Lys-Leu-Leu-Gln-Asp-Ile-Met-Ser-Arg-Gln-Gln-Gly-Glu-Ser-Asn-Gln-Glu-Arg-Gly-Ala-Arg-Ala-Arg-Leu-NH2 · MW 5135.91 g/mol · CAS 901758-09-6 · C221H366N72O67S

The one people reach for when they want to trim stubborn midsection fat and feel mentally sharp again - a premium, stabilized growth-hormone peptide, third-party verified and made in the USA.

Mechanism of action

Agonizes pituitary GHRH receptors with greater stability than endogenous GHRH.
Associated with triglyceride hydrolysis and lipolysis in adipocyte models.
Indirectly drives hepatic IGF-1 production.

Studied in the literature

Adipose-tissue lipolysis and metabolic-signaling models.
Hepatic lipid-metabolism signaling studies.
Cognitive-support modeling via GHRH signaling.

Selected citations

Falutz, J., et al. (2010). Effects of tesamorelin on visceral adipose tissue and metabolic parameters. New England Journal of Medicine. doi:10.1056/NEJMoa1006022 Stanley, T. L., et al. (2014). Effect of tesamorelin on visceral fat and liver fat: a randomized clinical trial. JAMA. doi:10.1001/jama.2014.8337

MOTS-cCellular energy, switched on

Met-Arg-Trp-Gln-Glu-Met-Gly-Tyr-Ile-Phe-Tyr-Pro-Arg-Lys-Leu-Arg · MW 2174.62 g/mol · CAS 1627580-64-6 · C101H152N28O22S2

Powered by your own mitochondria, MOTS-c is the favorite of athletes and biohackers chasing real endurance, steadier metabolism, and all-day energy - third-party verified, made in the USA, lot-traceable.

Mechanism of action

Triggers phosphorylation/activation of AMPK, a master energy regulator.
Promotes AICAR accumulation via folate-pathway regulation.
Promotes GLUT4 translocation linked to insulin-independent glucose uptake.

Studied in the literature

Insulin-sensitivity and metabolic-syndrome models.
Physical endurance and mitochondrial-biogenesis studies.
Cellular senescence and longevity modeling.

Selected citations

Lee, C., et al. (2015). The mitochondrial-derived peptide MOTS-c promotes metabolic homeostasis and prevents obesity and insulin resistance. Cell Metabolism. doi:10.1016/j.cmet.2015.01.009 Reynolds, J. C., et al. (2021). MOTS-c promotes physical performance and prevents age-associated metabolic decline. Nature Communications. doi:10.1038/s41467-020-20790-0

NAD+The longevity molecule

Coenzyme (dinucleotide) · MW 663.43 g/mol · CAS 53-84-9 · C21H27N7O14P2

The coenzyme at the center of the longevity movement - for the clear focus, deep energy, and youthful vitality you feel when your cells are firing on every cylinder - third-party verified, made in the USA, lot-traceable.

Mechanism of action

Key electron acceptor/donor in glycolysis and the Krebs cycle, fueling ATP production.
Mandatory co-substrate for Sirtuins (SIRT1-7) regulating mitochondrial biogenesis.
Substrate for PARP enzymes involved in DNA-strand-break repair.

Studied in the literature

Mitochondrial-respiration and aging models.
DNA-integrity and genotoxic-stress studies.
Neurodegenerative-disease modeling.

Selected citations

Gomes, A. P., et al. (Sinclair lab) (2013). Declining NAD+ induces a pseudohypoxic state disrupting nuclear-mitochondrial communication during aging. Cell. doi:10.1016/j.cell.2013.11.037 Yoshino, J., et al. (2018). NAD+ intermediates: the biology and therapeutic potential of NMN and NR. Cell Metabolism. doi:10.1016/j.cmet.2017.11.002

Research category

Metabolic & Incretin Signaling

01 peptides

GLP3-RTThe next-gen metabolic peptide

Tyr-Aib-Gln-Gly-Thr-Phe-Thr-Ser-Asp-Lys-Ser-Aib-Lys-Leu-Asp-Aib-Lys-Aib-Ala-Gln-Aib-Phe-Ile-Glu-Trp-Leu-Leu-Ala-Gly-Gly-Pro-Ser-Ser-Gly-Ala-Pro-Pro-Pro-Ser-NH2 · MW 4731.33 g/mol · CAS 2381089-83-2 · C223H343N53O66

Our most advanced metabolic peptide - a triple-pathway GLP-1/GIP/glucagon molecule built on the retatrutide structure for people serious about metabolic health and feeling their best - lab-tested purity, COA on every vial.

Mechanism of action

GLP-1R agonism associated with glucose-dependent insulinotropic signaling in vitro.
GIPR agonism synergizing insulinotropic signaling in receptor models.
GCGR agonism studied in hepatic and adipocyte energy-substrate signaling models.

Studied in the literature

GLP-1 / GIP / GCGR receptor-signaling and incretin-pathway studies.
Hepatocyte lipid-metabolism and incretin-signaling models.
Lipid-metabolism and vascular-signaling research models.

Selected citations

Coskun, T., et al. (2022). LY3437943, a novel triple GIP, GLP-1, and glucagon receptor agonist. Cell Metabolism. doi:10.1016/j.cmet.2022.07.013

Research category

Blends

02 peptides

GLOW BlendRecover, repair, glow - all in one

BPC-157 (10 mg) + TB-500 (10 mg) + GHK-Cu (50 mg)

Three powerhouse peptides in one vial - BPC-157, TB-500 and GHK-Cu working together for full-body recovery and skin that looks as good as you feel - lab-tested for purity, COA on every vial.

Mechanism of action

GHK-Cu drives collagen I/III transcription; TB-500 limits fibrosis via TGF-β.
BPC-157 (NO/VEGF) and TB-500 (endothelial migration) converge on angiogenesis.
TB-500 sequesters G-actin while BPC-157 raises GH receptors on migrated fibroblasts.

Studied in the literature

Musculoskeletal and myotendinous-junction repair models.
Complex cutaneous wound-healing models.
Hair-follicle remodeling studies.

Selected citations

Kajiya, K., et al. (2005). Thymosin beta 4 and VEGF display synergistic angiogenic properties in human dermal microvascular cells. Microvascular Research. doi:10.1016/j.mvr.2005.08.004

KLOW BlendOur most complete recovery stack

BPC-157 (10 mg) + TB-500 (10 mg) + GHK-Cu (50 mg) + KPV (10 mg)

Everything in our GLOW blend plus KPV - our most complete recovery stack, built for the days your body needs to bounce back fast and feel calm, comfortable, and ready again - third-party verified, made in the USA, lot-traceable.

Mechanism of action

KPV blocks NF-κB nuclear translocation, halting pro-inflammatory cytokine transcription.
KPV acts as a selective MC1R agonist, downregulating inflammatory cascades.
Reduced local inflammation lets GHK-Cu / BPC-157 rebuild tissue stability.

Studied in the literature

Severe dermatological inflammatory models (eczema, psoriasis).
High-inflammatory gastrointestinal recovery (colitis) models.
Hypertrophic-scar-resolution studies.

Selected citations

Kannengiesser, K., et al. (2008). Melanocortin tripeptide KPV suppresses NF-κB activation and IL-8 secretion in intestinal epithelial cells. Inflammatory Bowel Diseases. doi:10.1002/ibd.20302 Dalmasso, G., et al. (2008). PepT1-mediated tripeptide KPV uptake reduces intestinal inflammation. Gastroenterology. doi:10.1053/j.gastro.2007.11.026

Research category

Supplies

01 peptides

BAC WaterBacteriostatic Water

Bacteriostatic water for research use. Contains 0.9% benzyl alcohol.

Mechanism of action

Used for reconstituting lyophilized peptides.

Studied in the literature

Standard reconstitution agent for all peptide research.

Selected citations

Research Use Only

Every HappyTides peptide is made in the USA and characterized before it ships - lot-traceable, third-party tested, with a COA on every vial and purity confirmed before release. The fine print: products are sold for in-vitro laboratory research use only and are not intended to diagnose, treat, cure, or prevent any disease.

From the data to the vial

Every spec on this page ships in a vial.

Each reference compound is identity-verified, third-party tested, and traceable to its lot - with a scannable COA on every vial. For research use only.

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